Methods Immunophenotype was examined by flow cytometry in 31 patients with APL.
方法采用流式细胞术检测31例APL患者的免疫表型.
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Methods Analyzing clinical data of 18 patients with APL.
方法 分析18例APL患者的临床资料.
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Recently, similar cases have also been found in Caucasian APL.
近来在白种人APL中发现了具有t( 11;17)的病例.
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This is Jason Stone from APL Corporation.
我是APL公司 的贾森·斯通.
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The TM transcription and expression upregulated by ATRA may alleviate dysfunction of coagulation in APL.
ATRA增高TM转录及表达,缓解APL患者凝血功能异常.
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Method: The PML - RAR α fused protein in APL patients was stained by immuno - fluorescence antibody for PML.
方法: 采用免疫荧光技术检测APL患者PML-RARα融合蛋白.
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The positive rate of CD 15 in APL was significantly lower than those in CML - CP ( P & lt; 0.01 ).
有14.5%的APL患者 表达CD56,有48.2%的APL患者 表达CD15,CD15在APL的表达明显低于CML慢性 期( 48.2%vs95.2%,P & lt; 0.01 ).
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In contrast, APL cells of two cases showed synergistic differentiation after treatment with ATAR and bufalin.
为了进一步研究蟾蜍灵对ATRA诱导分化的增强效应, 我们研究蟾蜍灵与维甲酸联合对APL原 代培养细胞的诱导分化作用.
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Preclinical studies suggest that As 2 O 3 induces apoptosis and possibly differentiation in APL cells.
新近研究表明,As2O3不仅对APL细胞,而且对其它许多实体癌细胞均具有诱导凋亡作用.
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APL - wrapped version of the system from where the daily operation of bear out my own value.
APL被迫换版编制堵住在这些边际的常日运作证实了自己的代价.
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Objective : To detect the mechanism of hyperleukocytosis in patients with APL after treatment with ATRA.
目的: 揭示APL患者ATRA治疗后高白细胞征的机制.
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Methods : The clinical information of 62 patients with newly diagnosed APL was retrospectively analyzed.
方法回顾性分析62例初诊的APL患者的临床资料.
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IntroductionAll - trans retinoic acid ( ATRA ) is efficiently differentiation in - ducer for acute promyelocytic leukemia ( APL ).
全反式维甲酸 ( ATRA ) 用于急性早幼粒细胞白血病 ( APL) 的诱导分化治疗,有很高的完全缓解(CR ) 率,现已成为APL诱导缓解治疗的首选药物.
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Conclusion There are significant improvement in prognosis of APL treated with ATRA.
结论全反式维甲酸对急性早幼粒细胞性白血病患者的预后有很大改善.
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Objective To observe the clinical effects of all - trans - retinoic acid ( ATRA ) in treating 11 patients with acute promyelocytic leukemia ( APL ).
目的观察11例急性早幼粒细胞性白血病患者经全反式 维 甲酸 治疗后的临床效果.
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