ATPase was detected in the liver of Oncomelania snail.
钉螺肝脏具有三磷酸腺苷酶(ATPase)活力.
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If K + or Mg 2 was absent, gill Na - K - ATPase appears very low activity.
研究了牙鲜幼鱼由盐度3OpPt向低盐(24、18 、 12、6pPt)适应过程中血浆渗透压和鳃丝Na+一K+~ATPase活力的变化.
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The adenosine triphosphatease ( ATPase ) gene of turbot reddish body iridovirus ( TRBIV ) was cloned and sequenced.
克隆了大菱鲆红体病虹彩病毒 ( TRBIV ) 的重要功能蛋白———腺苷三磷酸酶(adenosinetriphosphatease,ATPase ) 基因的全部编码区及其上下游部分的核苷酸序列.
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Objective To research the changes of SDH and ATPase of expanded skeletal muscles in dogs.
目的:探讨骨骼肌扩张后琥珀酸脱氢酶(SDH)、三磷酸腺苷酶(ATPase)的变化.
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Objective To study the regularities of succinate dehydrogenase ( SDH ) and adenosine triphosphatase ( ATPase ) in myocardial cells with aging.
目的 探讨大鼠心肌细胞琥珀酸脱氢酶 ( SDH ) 和三磷酸腺苷酶 ( ATPase ) 的增龄变化规律.
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Previous research elucidated that ALR could interact with Na ~ +, K ~ + - ATPase directly in vitro and in vivo.
已有的研究表明,ALR与Na~+, K~+-ATPase在体内、体外都能直接结合.
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The vitamin K _ 3 - activated ATPase was inhibited by Amytal and Dicumarol.
维生素K_3对ATP酶活力的这种抑制作用受Amytal抑制.
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CONCLUSION: CL was a potent inhibitor of Ca 2 +, Mg 2 + ATPase.
结论: 马桑内酯是Ca2+, Mg2+?ATP酶的强力抑制剂.
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Thapsigargin is a highly selective inhibitor of the endoplasmic reticular Ca 2 + - dependent ATPase .
Thapsigargin是内质网Ca2 +-ATP 酶选择性抑制剂,可以不可逆地使内质网钙池排空,使胞质内Ca2+浓度持续升高.
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The activity of Na ~ + - K ~ + ATPase and Ca ~ ( 2 + ) - Mg ~ ( 2 + ) ATPase decreased.
黄芪组Na+-K+-ATP酶和Ca2 +-Mg2+ATP 酶活性均降低.
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Objective: Study the influence on rat brain edema and ATPase activity with cerebral ischemia reperfusion.
目的: 研究缺血再灌注对脑组织水肿和ATP酶活性的影响.
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Sequence Analysis on Genes Region From CO to ATPase 6 in mtDNA of Chinese Bombyx mandarina.
中国野桑蚕mtDNACO-ATPase6基因区域的序列测定及分析.
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Our results suggested that ACAD influenced the ATPase activiry of myosin through at least two pathways.
孔雀绿法测定肌球蛋白ATP酶 活性的结果显示:ACAD对 MLCK介导的肌球蛋白非钙依赖性磷酸化水平的提高与肌球蛋白ATP酶活性的增强呈线性关系.
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The ATPase decrease is closely correlative to the severity of the edema in peripheral nerves.
腺苷三磷酸酶活性下降与神经水肿密切相关.
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It may be one of the causes of lower Ca 2 + Mg 2 + ATPase activity.
这可能是导致AMI患者Ca2+-Mg2+-ATP酶活性降低的原因之一.
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