The expiratory ( F E ) and inspired ( F 1 ) concentration of isoflurane were monitored continuously.
连续监测吸入气 ( F1 ) 和呼出气 ( FB ) 中的异氟醚浓度.
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These inhibitory actions of isoflurane were not mediated by KATPchannels.
这些作用可能参与了异氟醚在体内的心脏保护作用,但异氟醚的这些有益作用并非由ATP钾通道介导.
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These patients may also benefit froma volatile anesthetic such as isoflurane that effectively bronchodilator the airways.
这些病人也可能使用吸入性麻醉剂,异氟烷可以有效的扩张气道.
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